Surgical Infectious DiseasesRichard J. Howard, Richard L. Simmons Aspen Publishers, 1988 - 913 pages |
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Page 221
... acids in sepsis , and because hepatic triglyceride production is increased , serum triglyceride and fatty acid levels are elevated . Fatty acids cleared by the liver are synthesized into triglycerides and ketone bodies . The glycerol ...
... acids in sepsis , and because hepatic triglyceride production is increased , serum triglyceride and fatty acid levels are elevated . Fatty acids cleared by the liver are synthesized into triglycerides and ketone bodies . The glycerol ...
Page 222
... acid . The result is a protein - based energy economy with muscle wasting and a progressively unbalanced amino acid pattern that the liver can no longer normalize . In such a setting , hepatic protein synthesis seems to become pro ...
... acid . The result is a protein - based energy economy with muscle wasting and a progressively unbalanced amino acid pattern that the liver can no longer normalize . In such a setting , hepatic protein synthesis seems to become pro ...
Page 240
... acid demonstrates accelerated metabolism and utilization of these substances following infection . 40 Amino acids move , via the bloodstream , from periph- eral protein stores to visceral organs where they are used for three fundamental ...
... acid demonstrates accelerated metabolism and utilization of these substances following infection . 40 Amino acids move , via the bloodstream , from periph- eral protein stores to visceral organs where they are used for three fundamental ...
Contents
Regional Surgical Infections 33 Infections of the Upper Gastrointestinal Tract 695 | 22 |
Enteric GramNegative Bacteria and Pseudo | 41 |
Anaerobic Bacteria | 50 |
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Surgical Infectious Diseases Richard L. Simmons,Richard J. Howard,Angela I. Henriksen Snippet view - 1982 |
Common terms and phrases
abscess acid activity acute addition aerobic agents amphotericin anaerobic animals antibiotic antibody antigen antimicrobial appear associated aureus bacteria become blood body cause cells changes chronic clinical colon combination common complement concentrations contaminated culture decreased defense demonstrated diagnosis direct disease doses drug effective factors fever flora fluid frequently function glucose Gram gram-negative growth hepatitis hospital host human Identification immune important increased indicated initial involved isolated laboratory lesions less levels macrophages major Manifestations mechanisms mixed normal occur operation oral organisms oxygen pathogenic patients penicillin percent pneumonia present produce protein pulmonary rare reactions reduced reported resistance respiratory response result role sepsis serum severe skin species specimens spread strains streptococci surface Surg Surgery surgical Table therapy tion tissue tract treated treatment types United urinary usually viral virus wound infection