Cancer Pain ManagementDeborah B. McGuire, Connie Henke Yarbro Grune & Stratton, 1987 - 286 pages |
From inside the book
Results 1-3 of 5
Page 3
... cells modulated the afferent impulses ( the " gate control " mechanism ) . Second , the afferent patterns in the fibers of the dorsal column acted as a central control trigger that activated selective brain processes . These processes ...
... cells modulated the afferent impulses ( the " gate control " mechanism ) . Second , the afferent patterns in the fibers of the dorsal column acted as a central control trigger that activated selective brain processes . These processes ...
Page 57
... cells are compromised by the chemotherapeutic drugs , with resultant thinning and ulceration of the mucosa . Oral trauma interacts with these processes to produce severe degradation of tissue . Oral mucositis occurs in more than 40 ...
... cells are compromised by the chemotherapeutic drugs , with resultant thinning and ulceration of the mucosa . Oral trauma interacts with these processes to produce severe degradation of tissue . Oral mucositis occurs in more than 40 ...
Page 167
... cells of the human body that contain microsomes . 103 Arachidonic acid , an essential fatty acid , exists as an inactive precursor of prostaglandin in the cell wall until molecular oxygen is incorporated into it by microcosmal enzymes ...
... cells of the human body that contain microsomes . 103 Arachidonic acid , an essential fatty acid , exists as an inactive precursor of prostaglandin in the cell wall until molecular oxygen is incorporated into it by microcosmal enzymes ...
Contents
Prevalence and Profile of Pain | 21 |
Painful Complications of Cancer Diagnosis | 47 |
Issues in Cancer Pain Management | 69 |
Copyright | |
8 other sections not shown
Other editions - View all
Common terms and phrases
achieved activity acute addition administration agents analgesia analgesic anxiety approach appropriate aspects aspirin assessment associated attitudes behavioral cancer pain cancer patients cancer-related pain cause changes chronic pain Clin clinical compared complete component considered continuous dependence depression described developed direct discussed disease dose drugs effects evaluation example experience experienced factors frequently function given important increased indicated individual infusion initial intensity interventions involved knowledge less major measures medication methods morphine narcotic narcotic analgesics nerve nurses observed occur oncology oral pain control pain management pain relief pain syndromes percent possible postoperative potential present problem procedures produce progressive psychological radiation received recent relatively relaxation reported respiratory response result scale scores sensory severe side effects significant specific symptoms Table techniques terminal therapy tolerance treated treatment tumor usually York